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Recent Progress in Epithelial-Mesenchymal Transitions Development - Cancer - Pathology (Cells Tissues Organs (Formerly Acta Anatomica)) by D. F Newgreen

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Published by S. Karger A. G. .
Written in English


  • Anatomy,
  • Medical,
  • Cell Differentiation,
  • Epithelium,
  • Mesoderm,
  • Physiology,
  • embryology

Book details:

The Physical Object
Number of Pages90
ID Numbers
Open LibraryOL12931778M
ISBN 103805579276
ISBN 109783805579278

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  During organogenesis, epithelial cells can give rise to mesenchymal cells through epithelial–mesenchymal transition. The reverse process, mesenchymal–epithelial transition (MET), can Cited by: Epithelial-Mesenchymal Transition. Epithelial–mesenchymal transition (EMT) is a bidirectional process whereby adherent epithelial cells gain migratory and invasive properties and vice versa and is thought to be important in the pathogenesis of CTCs, which express mesenchymal markers (Datta et . Epithelial-mesenchymal transition (EMT) is a type of epithelial plasticity that is characterized by long-lasting morphological and molecular changes in epithelial cells as a result of transdifferentiation towards a mesenchymal cell type. To detect possible phenotypic transitions in human cancer, surgical pathology is a useful medical discipline, examining surgical or biopsy material at the Cited by: Discover Book Depository's huge selection of W E Thompson books online. Free delivery worldwide on over 20 million titles.

Phase transitions in tumor growth VI: Epithelial-Mesenchymal transition Article in Physica A: Statistical Mechanics and its Applications February with 95 Reads How we measure 'reads'.   Cancer dissemination, which involves circulating tumor cells (CTCs), is a crucial step for metastasis. Recent progress in solid tumor stem cell biology suggests that cells that mediate metastasis are cancer stem cells. Invasive potential of cancer cells is induced by a process known as epithelial to mesenchymal transition (EMT).Cited by: 2. Recent work has suggested that there may be a linkage between the stem cell phenotype and that induced by the process of epithelial-mesenchymal transition (EMT). Notch signaling is a primitive signaling pathway having various roles in the normal origin and development of each multicellular organisms. Therefore, any aberration in the pathway will inevitably lead to deadly outcomes such as cancer. It has now been more than two decades since Notch was acknowledged as an oncogene in mouse mammary tumor virus-infected mice. Since that discovery, Cited by: 1.

ISBN: OCLC Number: Description: 1 online resource ( pages) Contents: Epithelial-mesenchymal transitions in development and tumor progression --Regulation of scatter factor (hepatocyte growth factor) production by tumor-stroma interaction --Regulation of HGF and HGFR gene expression --The role of scatter factor and the c-met proto-oncogene in angiogenic. Epithelial-mesenchymal transitions (EMTs) have been recently implicated in the process of cancer progression. The aim of this study was to assess how the preoperative expression patterns of EMT biomarkers correlate with the risk of postoperative invasion in ductal carcinoma in situ (DCIS) found on stereotactic breast biopsies. The book promotes cross-disciplinary communication between the sub-specialties of medicine. In common with the rest of the books in Translational Medicine, the book remains unified in theme by emphasizing recent innovations, critical barriers to progress, and the new tools being used to overcome them. Also includes specific areas of research. Lung Cancer Metastasis PDF Lung Cancer Metastasis PDF Free Download, Lung Cancer Metastasis PDF, Lung Cancer Metastasis Ebook Content Lung cancer is the leading cause of cancer-related mortality. Metastatic lung cancer is responsible for more than ninety percent of lung cancer related deaths. However, relatively little progress has been made in understanding the process.